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Depression medications "generally do more harm than good" say researchers

Research is showing that antidepressants do more harm than good and considerably shorten lifespan
Research is showing that antidepressants do more harm than good and considerably shorten lifespan
Frontiers in Psychology Journal

Doctors dispense prescriptions for them like sweets, yet research shows they damage your body...and the medical community has been ignoring the research.

A new study [1] has reviewed the research on the use and function of antidepressant medications and found that they "do more harm than good". In the abstract for the paper, the authors write that antidepressants damage your brain and nervous system cells, as reported previously. The authors describe how taking antidepressants causes neurons to become immature, which impairs how they function, and then results in their death.

Ask yourself this

If you are taking antidepressants right now, do you realize that you are paying money to buy pills that damage your own brain by killing your brain cells?

Taking antidepressant medications also cause several other problems. Which of these have you noticed?

  • Reduced libido, impaired mood, and the impact this has on a person's romantic and sexual life;[2]
  • The death of sperm cells [3], and its affect on a couple's fertility;
  • Digestive pain, diarrhea, constipation, indigestion, bloating, and headache; [4]
  • Increased bleeding during surgery[5], stroke[6, 7], and heart attack likelihood [8];
  • Reduced lifespan in the elderly; [1]
  • Increase the brain's susceptibility to future depressive episodes.[9]

Out of control
Even though they are initially prescribed to help people cope with temporary life challenges, antidepressant medications were meant to be used for around 3 months alongside therapy. The reality is that they end-up being prescribed for several months, and often years, which is when they damage the body.

Patients need to resolve what they feel depressed about by talking through what they need in order to let go of their stress and be at peace. In addition, with their physician's cooperation, patients can gradually lower the dose of their medication to nil over several weeks, because this is how long it takes to bring brain serotonin levels down for the body to be able to regulate itself again. [1].

According to the authors, when a person does quit these medications, they often find that symptoms of their depression continue until the body can naturally regulate its own serotonin levels. Their concern is that the patients may relapse while their physiology adjusts to living without the drugs. A solution to this is to ensure that other non-pharmacological solutions are in place, such as changing aspects of your lifestyle.

How the damage occurs
The authors' view is that these medications disrupt the brain and body's ability to naturally balance its serotonin system by taxing the bodily systems that regulate serotonin. They contend that from an evolutionary perspective, the use of these medications is unnatural and could "shorten the lifespan considerably."

If you presently take antidepressants, what are you going to do now that you know this information?

[1] Andrews, P. W., Thomson, J. A., Amstadter, A., & Neale, M. (2011). Primum non nocere: an evolutionary analysis of whether antidepressants do more harm than good. Frontiers in Psychology. doi: 10.3389/fpsyg.2012.00117

[2] Serretti, A., and Chiesa, A. (2009). Treatment-emergent sexual dysfunction related to antidepressants: a meta-analysis. J. Clin. Psychopharmacol. 29, 259–266.

[3] Tanrikut, C., Feldman, A. S., Altemus, M., Paduch, D. A., & Schlegel, P. N. (2010). Adverse effect of paroxetine on sperm. Fertil. Steril. 94, 1021–1026.

[4] Gershon, M. D. (2004). Serotonin receptors and transporters: roles in normal and abnormal gastrointestinal motility. Aliment. Pharmacol. Ther. 20, 3–14.

[5] van Haelst, I. M. M., Egberts, T. C. G., Doodeman, H. J., Traast, H. S., Burger, B. J., Kalkman, C. J., and van Klei, W. A. (2010). Use of serotonergic antidepressants and bleeding risk in orthopedic patients. Anesthesiology 112, 631–636.

[6] Smoller, J. W., Allison, M., Cochrane, B. B., Curb, J. D., Perlis, R. H., Robin- son, J. G., Rosal, M. C., Wenger, N. K., and Wassertheil-Smoller, S. (2009). Antidepressant use and risk of incident cardiovascular morbidity and mortality among postmenopausal women in the Women’s Health Initiative Study. Arch. Intern. Med. 169, 2128–2139.

[7] Trifiro, G., Dieleman, J., Sen, E. F., Gam- bassi, G., and Sturkenboom, M. C. J. M. (2010). Risk of ischemic stroke associated with antidepressant drug use in elderly persons. J. Clin. Psychopharmacol. 30, 252–258.

[8] Tata, L. J., West, J., Smith, C., Farrington, P., Card, T., Smeeth, L., and Hub- bard, R. (2005). General population based study of the impact of tri-cyclic and selective serotonin reuptake inhibitor antidepressants on the risk of acute myocardial infarction. Heart. 91, 465–471.

[9] Andrews, P. W., Kornstein, S. G., Halberstadt, L. J., Gardner, C. O., and Neale, M. C. (2011). Blue again: perturbational effects of antidepressants suggest monoaminergic homeostasis in major depression. Frontiers in Psychology. doi:10.3389/fpsyg.2011.00159