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The treatment goal for anyone with diabetes is to control blood sugar. Insulin is one of the most powerful drugs in our arsenal for this purpose....
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Find out more about Robert: Robert Scheinman joined the University of Colorado faculty in 1995. His laboratory has published on Diabetes, Arthritis, and Cancer. Robert teaches topics ranging from diabetes to chronic inflammatory disease to molecular biology. Email him: robscheinman@gmail.com |
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Diabetes Examiner's Articles
Showing: Treatment articles
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Byetta is a drug which stimulates insulin secretion while suppressing glucagon secretion. It works by stimulating a...
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Today the FDA indicated that the safety label of Byetta would be modified to include potential kidney problems...
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Rapid acting insulins are mutant forms of insulin that have been engineered to work faster upon injection than regular human insulins. The need for a...
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Carnitine has been of interest to diabetes doctors and researchers for some time. Indeed a search of the National Library of Medicine using the terms...
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We are focused these days on decreasing our environmental carbon footprint as a means to stabilize our planetary ecosystem. Similarly, our cells also...
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It helps to know what you are up against. Unfortunately, to study the lifetime effects of a chronic disease like diabetes takes…well it takes a...
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The cure for diabetes will involve replacing lost pancreatic function and what better source than your own stem cells. Getting from the stem cell to...
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Amylin Pharmaceuticals, along with Eli Lilly and Alkerms joined forces to set up a clinical trial comparing Lantus (insulin glargine) and Byetta...
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Sugar, as far a chemicals go, is pretty safe. No one calls in the hazmat team for spilled glucose in the laboratory. Sugar in your blood is a...
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Cheyenne Area Examiners:
- Depletion of cytosolic phospholipase A2 in bone marrow-derived macrophages protects against lung cancer progression and metastasis.
- Unmasking of a protective tumor necrosis factor receptor I-mediated signal in the collagen-induced arthritis model.
- Loss of a gimap/ian gene leads to activation of NF-kappaB through a MAPK-dependent pathway.
- AIRE recruits multiple transcriptional components to specific genomic regions through tethering to nuclear matrix.
- Partial activation precedes apoptotic death in T cells harboring an IAN gene mutation.
- Increased NF-kappa B activity in B cells and bone marrow-derived dendritic cells from NOD mice.
- The identification of a novel T cell activation state controlled by a diabetogenic gene.
